Dermatoheliosis

Dermatoheliosis

JOE R. MONROE, PA, MPAS
Dawkins Dermatology, Oklahoma City, Oklahoma

 

A lifetime of unprotected sun exposure had produced the following changes on the forearms of this 71-year-old rancher:

•Atrophy of epidermis and dermis, allowing the skin to tear and lacerate easily.

•The formation of multiple and, in this case, almost confluent actinic keratoses; these hyperkeratotic papules represent pre-cancerous focal atypia. Not only are these lesions uncomfortable, a small but significant percentage of them will undergo malignant transformation into squamous cell carcinoma.

•Solar purpura: these collections of trauma-induced extravasated red blood cells form very readily in such thin skin, but clear somewhat slowly for the same reason.

•Telangiectasias: these small red tortuous surface blood vessels are caused by a combination of chronic sun-induced vasodilatation, and thinning of the skin.

•Deep furrowing.

•Pronounced dryness.

•Mottled hyperpigmentary changes.

•Laxity/sagging, caused by the loss of dermal elasticity.

•Stellate scars and pseudoscars, which are common on these trauma-prone areas.

Dermatoheliosis

All of these skin changes represent dermatoheliosis.

For this patient, it is probably too late for sun protection, although it should be encouraged. At a minimum, he needs to be monitored regularly for skin cancer.

Treatment options for this patient include regular use of moisturizer and application of 5-fluorouracil cream to his arms twice a day for 3 weeks. The most prominent of the actinic keratoses could be treated with liquid nitrogen.